Clinical Utility of Patient-Derived Xenografts to Determine Biomarkers of Prognosis and Map Resistance Pathways in EGFR-Mutant Lung Adenocarcinoma.

Fiche publication


Date publication

août 2015

Journal

Journal of clinical oncology : official journal of the American Society of Clinical Oncology

Auteurs

Membres identifiés du Cancéropôle Est :
Pr MASCAUX Céline


Tous les auteurs :
Stewart EL, Mascaux C, Pham NA, Sakashita S, Sykes J, Kim L, Yanagawa N, Allo G, Ishizawa K, Wang D, Zhu CQ, Li M, Ng C, Liu N, Pintilie M, Martin P, John T, Jurisica I, Leighl NB, Neel BG, Waddell TK, Shepherd FA, Liu G, Tsao MS

Résumé

Although epidermal growth factor receptor (EGFR) -mutated adenocarcinomas initially have high response rates to EGFR tyrosine kinase inhibitors (TKIs), most patients eventually develop resistance. Patient-derived xenografts (PDXs) are considered preferred preclinical models to study the biology of patient tumors. EGFR-mutant PDX models may be valuable tools to study the biology of these tumors and to elucidate mechanisms of resistance to EGFR-targeted therapies.

Mots clés

Adenocarcinoma, metabolism, Adenocarcinoma of Lung, Afatinib, Aged, Aged, 80 and over, Animals, Antibodies, Monoclonal, Humanized, administration & dosage, Antineoplastic Agents, pharmacology, Biomarkers, Tumor, analysis, Carcinoma, Non-Small-Cell Lung, drug therapy, Cetuximab, Crizotinib, Drug Resistance, Neoplasm, ErbB Receptors, drug effects, Erlotinib Hydrochloride, Female, Gene Expression Profiling, Gene Expression Regulation, Neoplastic, Heterografts, Humans, Lung Neoplasms, drug therapy, Male, Mice, Mice, Inbred NOD, Mice, SCID, Middle Aged, Mutation, Predictive Value of Tests, Prognosis, Protein Kinase Inhibitors, pharmacology, Protein-Tyrosine Kinases, antagonists & inhibitors, Pyrazoles, administration & dosage, Pyridines, administration & dosage, Quinazolines, administration & dosage, Quinazolinones, administration & dosage, Signal Transduction, Up-Regulation

Référence

J. Clin. Oncol.. 2015 Aug 1;33(22):2472-80