Involvement of the ceramide signaling pathway in modulating the differentiated state of porcine thyroid cells.

Fiche publication


Date publication

novembre 2000

Journal

Lipids

Auteurs

Membres identifiés du Cancéropôle Est :
Pr MARTINY Laurent, Dr SCHNEIDER Christophe, Dr EL BTAOURI Hassan


Tous les auteurs :
Schneider C, Delorme N, Buisson-Legendre N, Bellon G, Emonard H, Btaouri HE, Hornebeck W, Haye B, Martiny L

Résumé

Neutral sphingomyelinase (Smase) is a cell membrane-associated phospholipase that hydrolyzes sphingomyelin to phosphocholine and ceramide, a lipid second messenger involved in cell differentiation and/or apoptosis. We first evidenced that porcine cultured thyroid cells could express neutral Smase activity even if thyrotropin (TStH), an essential hormone in thyroid cell differentiation, was found to induce a 1.7-fold decrease in Smase activity. Triggering the ceramide pathway by exogenous addition of neutral bacterial Smase (0.1 U/mL for 48 h), which transiently increased ceramide level by fourfold, drastically modified thyroid cell morphology. The follicle-like structures generated by TSH were disrupted, and the Smase-induced cell spreading was accompanied by a parallel loss of cell ability to iodinate proteins as well as a decrease of the adenylate cyclase system response. These inhibitory effects have been reproduced using short-chain exogenous ceramide analogs (C2-ceramides). Overall these data showed that ceramides emerged as potential mediators of dedifferentiation in thyroid cells.

Mots clés

Animals, Cell Differentiation, drug effects, Cells, Cultured, Ceramides, metabolism, Signal Transduction, Sphingomyelin Phosphodiesterase, metabolism, Swine, Thyroid Gland, cytology, Thyrotropin, pharmacology

Référence

Lipids. 2000 Nov;35(11):1259-68