Probing the Backbone Function of Tumor Targeting Peptides by an Amide-to-Triazole Substitution Strategy.

Fiche publication


Date publication

septembre 2015

Journal

Journal of medicinal chemistry

Auteurs

Membres identifiés du Cancéropôle Est :
Dr VALVERDE Ibai


Tous les auteurs :
Valverde IE, Vomstein S, Fischer CA, Mascarin A, Mindt TL

Résumé

Novel backbone-modified radiolabeled analogs based on the tumor targeting peptide bombesin were synthesized and fully evaluated in vitro and in vivo. We have recently introduced the use of 1,4-disubstituted 1,2,3-triazoles as metabolically stable trans-amide bond surrogates in radiolabeled peptides in order to improve their tumor targeting. As an extension of our approach, we now report several backbone-modified analogs of the studied bombesin peptide bearing multiple triazole substitutions. We investigated the effect of the modifications on several biological parameters including the internalization of the radiopeptidomimetics into tumor cells, their affinity toward the gastrin releasing peptide receptor (GRPr), metabolic stability in blood plasma, and biodistribution in mice bearing GRPr-expressing xenografts. The backbone-modified radiotracers exhibited a significantly increased resistance to proteolytic degradation. In addition, some of the radiopeptidomimetics retained a nanomolar affinity toward GRPr, resulting in an up to 2-fold increased tumor uptake in vivo in comparison to a (all amide bond) reference compound.

Mots clés

Amides, chemistry, Animals, Bombesin, analogs & derivatives, Cell Line, Tumor, Heterografts, Humans, Lutetium, Mice, Nude, Neoplasm Transplantation, Peptide Fragments, chemistry, Peptidomimetics, chemistry, Proteolysis, Radiopharmaceuticals, chemistry, Receptors, Bombesin, metabolism, Structure-Activity Relationship, Tissue Distribution, Triazoles, chemistry

Référence

J. Med. Chem.. 2015 Sep 24;58(18):7475-84